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Bioessays impact factor 2011

Marta C. Guadamillas, Ana Cerezo, Miguel A. J Cell Sci 1 October ; 19 : — Occasionally, detached or misplaced cells can overcome anoikis and survive for a certain period of time in the absence of the correct signals from the extracellular matrix ECM.

Elsevier Announces 2011 Journal Impact Factor Highlights

Journal Impact Factor | Impact Factor List | | |

Human epidermal growth factor receptor 2 HER2 is a member of the epidermal growth factor receptor family having tyrosine kinase activity. Dimerization of the receptor results in the autophosphorylation of tyrosine residues within the cytoplasmic domain of the receptors and initiates a variety of signaling pathways leading to cell proliferation and tumorigenesis. HER2 overexpression has also been seen in other cancers like ovary, endometrium, bladder, lung, colon, and head and neck. The human epidermal growth factor receptor HER family of receptors plays a central role in the pathogenesis of several human cancers. They regulate cell growth, survival, and differentiation via multiple signal transduction pathways and participate in cellular proliferation and differentiation.

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The following is a partial list of scientific journals. There are thousands of scientific journals in publication, and many more have been published at various points in the past. The list given here is far from exhaustive, only containing some of the most influential, currently publishing journals in each field. As a rule of thumb, each field should be represented by fewer than ten positions, chosen by their impact factors and other ratings. Note : there are many science magazines that are not scientific journals, including Scientific American , New Scientist , Australasian Science and others.
Akt1 is involved in cellular survival pathways, by inhibiting apoptotic processes. Akt1 is also able to induce protein synthesis pathways, and is therefore a key signaling protein in the cellular pathways that lead to skeletal muscle hypertrophy, and general tissue growth. Mouse model with complete deletion of Akt1 manifests growth retardation and increased spontaneous apoptosis in tissues such as testes and thymus. Akt now also called Akt1 was originally identified as the oncogene in the transforming retrovirus , AKT8.

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